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Myocardial Infarction: Difference between revisions
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==Treatment== | ==Treatment== | ||
[[File:Figure_2_-_Reperfusion_strategies.svg|thumb| | [[File:Figure_2_-_Reperfusion_strategies.svg|thumb|right|500px|Reperfusion strategies. The thick arrow indicates the preferred strategy.]] | ||
As the formation of an intracoronary thrombus is a central mechanism in ACS and (recurrent) subsequent outcomes, the cornerstone in the treatment of ACS is antithrombotic treatment. All patients diagnosed with ACS should start with aspirin and a P2Y12 receptor blocker (clopidogrel, prasugrel or ticagrelor). <Cite>REFNAME15</Cite> Aspirin and the P2Y12 receptor blocker are both platelet aggregation inhibitors. The treatment of ACS also focuses on medication to reduce the workload of the heart. ß blockers lower heart rate and blood pressure, to decrease the oxygen demand of the heart. <Cite>REFNAME16</Cite> Nitrates dilatate the coronary arteries. <Cite>REFNAME17</Cite> | As the formation of an intracoronary thrombus is a central mechanism in ACS and (recurrent) subsequent outcomes, the cornerstone in the treatment of ACS is antithrombotic treatment. All patients diagnosed with ACS should start with aspirin and a P2Y12 receptor blocker (clopidogrel, prasugrel or ticagrelor). <Cite>REFNAME15</Cite> Aspirin and the P2Y12 receptor blocker are both platelet aggregation inhibitors. The treatment of ACS also focuses on medication to reduce the workload of the heart. ß blockers lower heart rate and blood pressure, to decrease the oxygen demand of the heart. <Cite>REFNAME16</Cite> Nitrates dilatate the coronary arteries. <Cite>REFNAME17</Cite> | ||